Motor Movement Cortex and Damage Causing Disorders. Part 3
Motor disorders can be divided into two principal groups, hyperkinesia (over-activity) and hypo-kenesia, too little movement. The former is about a wide group of disorders that are involuntary muscle movements, show shaking of various body parts, tics which are uncontrollable and rapid, disjointed movements and or sounds.
Sudden shock-like muscle contractions are symptoms of myo-clonus, wide, quick random and jerky movements and caused by chorea and ballism. Hyper-kinesia disorders are generally slowness in movements (brady-kinesia) “freezing” or inability to begin a movement or involuntary arrest of a movement; rigidity is increased muscle tension when a limb encounters force and postural instability which is the loss of ability to stand up straight in an upright position.
In the Primary motor cortex damage may cause paralysis or a weakness of the opposite side of the lesion, the lesions in this case Motor muscle Cortex areas, and The Parietal Cortex may cause damage of distance of position in space or speed of objects. The Supplementary Cortex (area 4) may cause prevention of the planning of movements “blocked” pathways from here to motor cortex may cause forms of paralysis. The Mid-brain damage may cause tics, or block voluntary movements, injury to substantia nigra in mid-brain area reduces ability to initiate movement. The damage to the Cerebellum injury can prevent fine timing of movements and can cause tremors. The damage to a spinal cord can cause paralysis and a loss of motor control and cause spascisity. Areas affected in much of a brain involved with movement and so many different brain injuries lead to serious motor disorders and sometimes death. If you have a precisely timed sequence of motor neuronal patterned firings, these firings when damaged can cause a lot of muscle damage when interrupted by some virus such as a protein miss-folding, and causing nuclei destabilization by Covid 19. The axon of the motor neuron between the spinal vertebrae travel to nerve endings infiltrating muscle fiber at neuromuscular junctions, and when these neurotransmitters fire they release a neurotransmitter called acetycholine. This diffuses across the narrow “synaptic cleft,” connecting the muscle to the nerve and binds to acetycholine receptors in the muscle cell membrane, which by a series of reactions makes the specific muscle contract. Muscles that are required to carry out fine movements have correspondingly higher numbers of neurons than those required to preform gross movements. When stimulated by a MOTOR NERVE, electrical changes cause the release of calcium ions inside the muscle. This causes the filaments of the muscles to slide against each other and contract.
The miracle of our muscles, our cortex groups of neuronal passageways, and neuronal synaptics and pathways, play a huge part in any movement and change in our body. In the case of those that were subjected to this virus, the nuclei being destabilized is hardly rocket science and would be seen very clearly by those who do Medical treatments to patients as covid 19 was obvious. I want to thank Dr. Hansen for his integrity and showing the autopsies of cortex damage to the grey and white matter in the cortex and other systems down the CNS, the heart, the kidneys, the lungs, the lung lining, and those who know of nose damage, and mold, taste damage, oxygen depletion in blood coagulating, and in thrombosis. It becomes every so clear why Myocarditis, Pericarditis, Respiratory disease is of great concern for the children, and this leaves our proteins vulnerable to miss-folding processes in the future. When proteins miss-fold, and cause the nuclei to destabilize the hereditary genetics that could cause serious illness such as muscle diseases, or Frontal Temporal Disorders are significantly showing up in our populations around the world from Covid 19 and all variations. The damage to the nuclei shows that the virus is doing damage to the DNA and the mRNA and proteins as it is obvious it has damaged the nuclei. Carolyn d Hogarth